OncoMatch

OncoMatch/Leukemia — Chronic Myeloid (CML)/ABL1 kinase domain mutation

Leukemia — Chronic Myeloid (CML)ABL1 kinase domain mutation Clinical Trials

18 recruiting trials·Updated daily from ClinicalTrials.gov

ABL1 kinase domain mutations — most notably T315I — are the primary mechanism of TKI resistance in CML, conferring varying degrees of resistance to first-, second-, and third-generation inhibitors. T315I is resistant to most ATP-competitive TKIs and is targetable with ponatinib and asciminib (STAMP inhibitor), though compound mutations can limit activity. Trials investigate mutation-specific treatment sequencing, asciminib combinations, and novel allosteric ABL1 inhibitors.

Match trials to my profileClinician mode →
Additional trials

More ABL1 kinase domain mutation Leukemia — Chronic Myeloid (CML) trials

Phase 2
Phase 1
Phase 4 / Other
6 international trials (no US sites)
Other Leukemia — Chronic Myeloid (CML) biomarkers

Browse other molecular targets with active Leukemia — Chronic Myeloid (CML) trials.

BCR::ABL1