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OncoMatch/Clinical Trials/NCT05327010

Testing the Combination of the Anti-cancer Drugs ZEN003694 (ZEN-3694) and Talazoparib in Patients With Advanced Solid Tumors, The ComBET Trial

Is NCT05327010 recruiting? Yes, currently enrolling (May 2026). This Phase 2 trial studies multiple treatments including BET Bromodomain Inhibitor ZEN-3694 and Talazoparib for advanced malignant solid neoplasm.

Phase 2RecruitingNational Cancer Institute (NCI)NCT05327010Data as of May 2026

Treatment: BET Bromodomain Inhibitor ZEN-3694 · TalazoparibThis phase II trial tests whether ZEN003694 (ZEN-3694) in combination with talazoparib works to shrink tumors in patients with solid tumors that are unlikely to be cured or controlled with treatment and that may have spread from where it first started to nearby tissue, lymph nodes, or distant parts of the body (advanced). Another aim of this study is to find out if, and how, patients' genes influence their response to this specific drug combination. For this part of the study, investigators will run tests using samples of patients' tumor tissue and blood that will be collected during the study. ZEN-3694 is an inhibitor of a family of proteins called the bromodomain and extra-terminal (BET). It may prevent the growth of tumor cells that overproduce BET protein. Talazoparib is an inhibitor of PARP, an enzyme that helps repair deoxyribonucleic acid (DNA) when it becomes damaged. Blocking PARP may help keep cancer cells from repairing their damaged DNA, causing them to die. PARP inhibitors are a type of targeted therapy. Genes are pieces of the DNA code that individuals inherit from their parents. Some genes work to protect against cancer by correcting damage that can occur in the DNA when cells divide. BRCA1 and BRCA2 are two examples of these types of genes, and they are called tumor-suppressor genes. For example, if a person has a mutation in a BRCA1/2 gene they have a greatly increased risk of developing breast and ovarian cancer because their cells may no longer be able to completely repair damaged DNA. It is the accumulation of DNA damage which causes a cell to change into a cancerous cell. Other genes are also involved in this process, and these are called DNA damage repair genes. The KRAS mutation is a change in a protein in normal cells. Normally KRAS serves as an information hub for signals in the cell that lead to cell growth, but when there is a mutation in KRAS it signals too much and cells grow without being told to, which causes cancer. Combination therapy with ZEN-3694 and talazoparib may be effective at slowing or stopping tumor growth in patients with advanced cancer.

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Extracted eligibility criteria

Cancer type

Tumor Agnostic

Biomarker criteria

Required: BRCA1 germline or somatic mutation

Cohort 1 must have (i) a germline or somatic mutation in BRCA1

Required: BRCA2 germline or somatic mutation

Cohort 1 must have (i) a germline or somatic mutation in BRCA2

Required: BARD1 germline or somatic mutation

Cohort 2 must have: (i) a germline or somatic mutation in any of the following deoxyribonucleic acid (DNA) damage response (DDR) genes: BARD1

Required: FANCA germline or somatic mutation

Cohort 2 must have: (i) a germline or somatic mutation in any of the following deoxyribonucleic acid (DNA) damage response (DDR) genes: FANCA

Required: BRIP1 germline or somatic mutation

Cohort 2 must have: (i) a germline or somatic mutation in any of the following deoxyribonucleic acid (DNA) damage response (DDR) genes: BRIP1

Required: PALB2 germline or somatic mutation

Cohort 2 must have: (i) a germline or somatic mutation in any of the following deoxyribonucleic acid (DNA) damage response (DDR) genes: PALB2

Required: RAD51 germline or somatic mutation

Cohort 2 must have: (i) a germline or somatic mutation in any of the following deoxyribonucleic acid (DNA) damage response (DDR) genes: RAD51

Required: RAD51C germline or somatic mutation

Cohort 2 must have: (i) a germline or somatic mutation in any of the following deoxyribonucleic acid (DNA) damage response (DDR) genes: RAD51C

Required: RAD51D germline or somatic mutation

Cohort 2 must have: (i) a germline or somatic mutation in any of the following deoxyribonucleic acid (DNA) damage response (DDR) genes: RAD51D

Required: KRAS mutation

Cohort 4 must have KRAS mutated advanced solid tumors

Allowed: KRAS G12C

Patients with KRAS G12C mutations must have already had KRAS G12C targeted therapy (e.g., sotorasib) previously

Disease stage

Metastatic disease required

metastatic or unresectable and ... at least one measurable disease site, as defined by Response Evaluation Criteria in Solid Tumors (RECIST) version (v) 1.1

Performance status

ECOG 0–2(Ambulatory, capable of self-care)

Prior therapy

Min 1 prior line

Must have received: PARP inhibitor

must have received prior PARPi monotherapy or PARPi combination-therapy

Cannot have received: ZEN003694 (ZEN-3694) or investigational BET inhibitor (ZEN003694, ZEN-3694)

Patients who have previously received ZEN003694 (ZEN-3694) or who have been treated with an investigational BET inhibitor

Lab requirements

Blood counts

Absolute neutrophil count >= 1,500/mcL; Platelets >= 150,000/mcL; Hemoglobin >= 10.0 g/dL (no blood transfusions in the preceding 28 days)

Kidney function

Creatinine <= 1.5 x institutional ULN OR GFR >= 60 mL/min/1.73 m^2 for subjects with creatinine levels > 1.5 x institutional ULN, unless data exists supporting safe use at lower kidney function values, no lower than 30 mL/min/1.73 m^2

Liver function

Total bilirubin <= 1.5 x institutional ULN OR direct bilirubin = ULN for subjects with total bilirubin levels > 1.5 x ULN; AST/ALT <= 2.5 x institutional ULN

Cardiac function

NYHA class 2B or better

Absolute neutrophil count >= 1,500/mcL; Platelets >= 150,000/mcL; Hemoglobin >= 10.0 g/dL (no blood transfusions in the preceding 28 days); Total bilirubin <= 1.5 x institutional ULN OR direct bilirubin = ULN for subjects with total bilirubin levels > 1.5 x ULN; AST/ALT <= 2.5 x institutional ULN; Creatinine <= 1.5 x institutional ULN OR GFR >= 60 mL/min/1.73 m^2 ... NYHA class 2B or better

Structured fields extracted by AI. May contain errors — verify against the official protocol.

US trial sites

  • City of Hope Comprehensive Cancer Center · Duarte, California
  • City of Hope at Irvine Lennar · Irvine, California
  • UC San Diego Moores Cancer Center · La Jolla, California
  • Keck Medicine of USC Koreatown · Los Angeles, California
  • Los Angeles General Medical Center · Los Angeles, California

Showing up to 5 US sites. See all sites on ClinicalTrials.gov →

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