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OncoMatch/Clinical Trials/NCT03852407

Allogeneic Hematopoietic Cell Transplantation From HLA-matched Donor After Flu-Mel-PTCy Versus Flu-Mel-ATG Reduced-intensity Conditioning

Is NCT03852407 recruiting? Yes, currently enrolling (May 2026). This Phase 2 trial studies multiple treatments including Thymoglobulin and Melphalan for acute myeloid leukemia in remission.

Phase 2RecruitingUniversity of LiegeNCT03852407Data as of May 2026

Treatment: Thymoglobulin · Melphalan · Fludarabine · CyclophosphamidThe present project aims at comparing two conditioning regimens (FM-PTCy vs FM-ATG). The hypothesis is that one or the two regimens will lead to a 2-year cGRFS rate improvement from 30% (the cGRFS rate with FM without ATG/PTCy) to 45% (Pick-a-winner phase 2 randomized study).

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Extracted eligibility criteria

Cancer type

Acute Myeloid Leukemia

Myelodysplastic Syndrome

Chronic Myeloid Leukemia

Myeloproliferative Neoplasm

Acute Lymphoblastic Leukemia

Multiple Myeloma

Hodgkin Lymphoma

Non-Hodgkin Lymphoma

Lab requirements

Liver function

No fulminant liver failure, cirrhosis with portal hypertension, alcoholic hepatitis, esophageal varices, history of bleeding esophageal varices, hepatic encephalopathy, uncorrectable hepatic synthetic dysfunction evinced by prolongation of the prothrombin time, ascites related to portal hypertension, bacterial or fungal liver abscess, biliary obstruction, chronic viral hepatitis with total serum bilirubin >3 mg/dL, or symptomatic biliary disease

Cardiac function

No symptomatic coronary artery disease; ejection fraction <40%; uncontrolled arrhythmia, uncontrolled hypertension

Terminal organ failure, except for renal failure (dialysis acceptable) 1. Cardiac: Symptomatic coronary artery disease; ejection fraction <40%; uncontrolled arrhythmia, uncontrolled hypertension; 2. Pulmonary: DLCO< 40% and/or receiving supplementary continuous oxygen, FEV1< 40%; 3. Hepatic: Fulminant liver failure, cirrhosis of the liver with evidence of portal hypertension, alcoholic hepatitis, esophageal varices, a history of bleeding esophageal varices, hepatic encephalopathy, uncorrectable hepatic synthetic dysfunction evinced by prolongation of the prothrombin time, ascites related to portal hypertension, bacterial or fungal liver abscess, biliary obstruction, chronic viral hepatitis with total serum bilirubin >3 mg/dL, and symptomatic biliary disease

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